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Evidence supporting the causal relationship of both passive and active exposure to tobacco smoke with the development of atherosclerosis and cardiovascular morbidity, beginning as early as fetal life, has been found in a wide array of studies, including those using direct measurement of atherosclerosis in humans and animals, noninvasive measurement of injury to cardiovascular end organs, and measurement of associations with biomarkers known to be associated with atherosclerosis and other forms of cardiovascular disease

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Not to sound morbid, but eventually existing smokers will die, and then what

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