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Mitochondrial and fatty changes in hepatocytes of patients with Wilsons disease

We have already observed that BL patients have weaker correlations within the TRP pathway, e.g., 5-HIAA with TRP (tau = 0.09 BL, tau = 0.69 HC), which may occur when dietary associations are overcome by other rate-limiting pathway controls based on physiological needs for serotonin neurotransmitter (Yao et al., 2010a)

Fortunately, oral supplementation plays a crucial role in overcoming this deficiency and can usually get things back on track

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However, the frequent occurrence of compensated activation of alternative pathways prominently hinders the efficacy of targeting one single metabolic paradigm