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Previously, we have shown that chemogenetic activation of GIPR-expressing cells in either the hypothalamus or the hindbrain supresses food intake 7,8 , and GIPR-expression in oligodendrocytes has recently been linked to enhanced access of co-administered GLP-1R agonists 23

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doi: 10.3390/ijms241914857 32

The activation of GLP-1R triggers a complex intracellular signaling cascade, which at the end activates protein kinase A (PKA) pathway via production of cyclic adenosine monophosphate (cAMP) ( GLP-1 exerts potent glucose-lowering effects through multiple mechanisms including insulin release stimulation and suppression of glucagon secretion

This subset of diagnoses is not only large (and growing) but affects a disproportionately large portion of the overall population, and accounts for an even larger portion of morbidity and overall mortality