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Although IDH is a potential source of NADPH, its major function is irrelevant to lipogenesis [172]

Hematocrit, lipids, glucose tolerance, and liver enzymes warrant closest attention in dual therapy patients

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Some individuals are genetically predisposed to robust GLP-1 signaling, while others show blunted responses

In agreement with this latter finding, an increase in thromboxane synthesis was described in homocystinuric patients (Di Minno et al, 1993)