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Clinical Pharmacology and Therapeutics, 85(3), 296304

In PTSD mice models, trauma exposure increased tyrosine hydroxylase expression (the rate-limiting enzyme in catecholamine biosynthesis), elevating stress hormones epinephrine and norepinephrine in serum and colon, which upregulated claudin-2 expression, confirming gut barrier dysfunction (Yadav et al., 2023)

Whether seeking injury recovery, muscle growth, gut health improvements, or general anti-inflammatory advantages, scheduling should be aligned with individual goals

It supports the study of combined effects, optimal dosing for different injury types, timing protocols for acute versus chronic cases, and the link between reduced inflammation and tissue quality

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