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As expected, CH&Desmo inhibited RSL3 induced cell death, and SQLE overexpression largely restored ferroptosis protection, which was further strengthened by FDFT1 overexpression, comparable to WT cell level (Fig
Meanwhile, the phosphorylation level of p38 MAPK was significantly elevated, and its ferroptosis inhibitors, SB202190, reduced ROS production, restored the expression of ferroptosis - related genes, and promoted cell survival by blocking this pathway
& McGavock, J
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