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4,11,15 Inflammatory responses mediated by proinflammatory cytokines and pathways, such as nuclear factor kappa B (NF-B) and Toll-like receptor 4 (TLR4), worsen the condition by amplifying oxidative stress and hindering neuronal repair

For example, investigating the impact of GSH on insulin sensitivity, lipid metabolism, and pro-inflammatory cytokine levels could provide valuable insights into its role as a metabolic therapy

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