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Furthermore, a recent study in human subjects showed a significant association between quartiles of serum homocysteine levels and coronary microvascular endothelial dysfunction in patients with early coronary atherosclerosis [88], confirming the detrimental effect of homocysteine on endothelial function

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NO generated by iNOS can induce DNA damage through the formation of peroxynitrite (ONOO ), a reactive nitrogen species that can cause mutagenesis and genomic instability, thus driving tumor progression (60)

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